Volume 12, Number 12—December 2006
Research
Influenza, Campylobacter and Mycoplasma Infections, and Hospital Admissions for Guillain-Barré Syndrome, England
Clarence C. Tam*†
, Sarah J. O’Brien‡, and Laura C. Rodrigues*
, Sarah J. O’Brien‡, and Laura C. Rodrigues*
Table 3
Poisson regression of Guillain-Barré syndrome (GBS) admissions against infection reports, England, 1993–2002: unclustered lags*
| Pathogen | All ages | <35 y | 35–64 y | >65 y |
|---|---|---|---|---|
| Campylobacter | – | 7 | – | – |
| Influenza | – | – | R3 | – |
| A | – | – | R1,1 | – |
| B | – | – | 2,5 | – |
| Myplasma pneumoniae | – | 3 | – | 2 |
| Haemophilus influenzae non-B | 8 | – | 7 | – |
| H. influenzae B | – | 5 | – | R2 |
| Cytomegalovirus | – | R3,5 | – | 6 |
| Epstein-Barr virus | – | R1 | 5 | – *Lags significant at the 0.05 level of precision but not occurring in clusters (regression models are adjusted for yearly trend, seasonal pattern [up to 6th harmonic] and public holidays). *Lags significant at the 0.05 level of precision but not occurring in clusters (regression models are adjusted for yearly trend, seasonal pattern [up to 6th harmonic] and public holidays). |
*Lags significant at the 0.05 level of precision but not occurring in clusters (regression models are adjusted for yearly trend, seasonal pattern [up to 6th harmonic] and public holidays).
Page created: October 04, 2011
Page updated: October 04, 2011
Page reviewed: October 04, 2011
The conclusions, findings, and opinions expressed by authors contributing to this journal do not necessarily reflect the official position of the U.S. Department of Health and Human Services, the Public Health Service, the Centers for Disease Control and Prevention, or the authors' affiliated institutions. Use of trade names is for identification only and does not imply endorsement by any of the groups named above.