Molecular Epidemiology of Influenza A(H1N1)pdm09 Virus among Humans and Swine, Sri Lanka
Harsha K.K. Perera, Dhanasekaran Vijaykrishna, Akuratiya G. Premarathna, Chrishan J.S. Jayamaha, Geethani Wickramasinghe, Chung L. Cheung, Ming F. Yeung, Leo L.M. Poon, Aluthgama K.C. Perera, Ian Barr, Yi Guan, and Ahmad M. Hakawi
Author affiliations: University of Hong Kong, Hong Kong, China (H.K.K. Perera, C.L. Cheung, M.F. Yeung. L.L.M. Poon, Y. Guan, M. Peiris); University of Kelaniya, Kelaniya, Sri Lanka (H.K.K. Perera, A.G. Premarathna); Duke-National University of Singapore Graduate Medical School, Singapore (D. Vijaykrishna); Medical Research Institute, Colombo, Sri Lanka (C.J.S. Jayamaha, G. Wickramasinghe); Colombo Municipal Council, Colombo (A.K.-C. Perera); WHO Collaborating Centre for Reference and Research on Influenza, Melbourne, Australia (I.G.Barr)
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Figure 1
Figure 1.
Phylogenetic relationships of influenza A H1N1pdm viruses isolated from human and swine during 2009–2012 in Sri Lanka. (A) Maximum likelihood tree generated from a concatenated dataset of 8 gene segment sequences from 1,057 human and 82 swine H1N1pdm viruses isolated globally during 2009–2012, and 35 human and 26 swine H1N1pdm viruses isolated in Sri Lanka in 2009–2012. Red and green branches represent human (hu) and swine (sw) viruses isolated in Sri Lanka, respectively; gray and blue branches represent globally sampled human and swine viruses, respectively. Highlighted regions (gray) are shown with virus names in (B) and (C), respectively. Support values estimated from 500 maximum likelihood bootstrap replicates are shown along the node for each swine and human clusters identified in Sri Lanka in (A) and for each node with >70% support for (B) and (C).
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