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Volume 23, Number 2—February 2017

Spread and Evolution of Respiratory Syncytial Virus A Genotype ON1, Coastal Kenya, 2010–2015

James R. OtienoComments to Author , Everlyn M. Kamau, Charles N. Agoti, Clement Lewa, Grieven Otieno, Ann Bett, Mwanajuma Ngama, Patricia A. Cane, and D. James Nokes
Author affiliations: Kenya Medical Research Institute (KEMRI)–Wellcome Trust Research Programme, Kilifi, Kenya (J.R. Otieno, E.M. Kamau, C.N. Agoti, C. Lewa, G. Otieno, A. Bett, M. Ngama, D.J. Nokes); Pwani University, Kilifi (C.N. Agoti); Public Health England, Salisbury, UK (P.A. Cane); University of Warwick, Coventry, UK (D.J. Nokes)

Main Article

Table 2

Clinical severity comparison between cases of pneumonia caused by respiratory syncytial virus A genotypes ON1 and GA2 in children admitted to Kilifi County Hospital, September 2010–August 2015*

Characteristic Unadjusted odds ratio (95% CI) p value
Age <1 y 0.85 (0.49–1.49) 0.579
Male sex
0.76 (0.52–1.12)
Clinical features
Cough 1.16 (0.31–4.37) 0.830
Breathing difficulty 0.28 (0.08–0.97) 0.045
Chest wall indrawing 1.46 (0.46–4.59) 0.520
Inability to feed 2.40 (1.31–4.36) 0.004
Oxygen saturation <90% 0.86 (0.53–1.38) 0.521
1.67 (0.75–3.74)
Pneumonia status, very severe 0.89 (0.57–1.39) 0.609
Hospital stay, >4 d 0.77 (0.53–1.14) 0.192
Outcome, died 1.416 (0.42–4.78) 0.575

*The denominator was 442 samples but for some of the features it ranged from 439 to 442.

Main Article

Page created: January 17, 2017
Page updated: January 17, 2017
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