Asymmetric Type F Botulism with Cranial Nerve Demyelination
Alina Filozov1 , Jessica A. Kattan1, Lavanya Jitendranath, C. Gregory Smith, Carolina Lúquez, Quyen N. Phan, and Ryan P. Fagan
Author affiliations: Middlesex Hospital, Middletown, Connecticut, USA (A. Filozov, L. Jitendranath); Centers for Disease Control and Prevention, Atlanta, Georgia, USA (J.A. Kattan, C. Lúquez, R.P. Fagan); Connecticut Department of Public Health, Hartford, Connecticut, USA (J.A. Kattan, Q.N. Phan); North Carolina Department of Health and Human Services, Raleigh, North Carolina, USA (C.G. Smith)
Figure. Postmortem cranial nerve tissue from a patient with botulism. A) Fragmentation of myelin sheaths and inflammatory infiltration of B and CD3+ T-cells within the nerve tissue (original magnification ×200). B) B-cell infiltration of nerve tissue; C) CD68-positive myelinoklastic macrophages (original magnification ×400).
1These authors contributed equally to this article.
Page created: December 20, 2011
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